FINLÂNDIA
FINLANDIA
Reencontrei (07/08/2022) o buquê que ela tinha na mão. Imagem já publicada no AMICOR 3.074 do meio do mês de março do corrente ano.
King's Williams Island Finland 1987 - colhendo flores silvestres...
Endomyocardial fibrosis remains an important cause of restrictive cardiomyopathy despite the unsolved questions regarding the cause and therapeutic strategies. Worldwide prevalence is estimated at 10 to 12 million in 2008.1 Echocardiography is the standard modality for endomyocardial fibrosis diagnosis. Ventricular endocardial fibrosis with organized thrombus is the hallmark of advanced disease.2
In this case, a 70-year-old male patient was admitted with symptoms of right heart failure. ECG demonstrated atrial fibrillation and right bundle-branch block. Transthoracic, 2-dimensional, and 3-dimensional transesophageal echocardiography evidenced obliteration of the right ventricular (RV) apex, severe right atrial enlargement with a prominent aneurysm of fossa ovalis, and inferior vena cava dilatation (Figure, Movies I and II in the Data Supplement). Myocardial contrast echocardiography revealed marked RV apex and subtle left ventricular apex subendocardial delayed perfusion and a small perfusion defect over the RV endocardium (Figure, Movie III in the Data Supplement). Late gadolinium enhancement cardiac magnetic resonance imaging showed mild RV systolic dysfunction, apical thickening and obliteration, hypoperfusion at rest, and typical late double V enhancement, compatible with subendocardial fibrosis and thrombus (Figure, Movie IV in the Data Supplement). Left ventricular early involvement was demonstrated by the presence of hypoperfusion at rest and late subendocardial enhancement in its apex and apical lateral segment, as well as involvement of the mitral valve (Figure, Movie IV in the Data Supplement).

Figure. Echocardiography and cardiac magnetic resonance (CMR) images.A, Transthoracic echocardiography and (B) Transesophageal echocardiography (TEE) evidenced obliteration of the right ventricular (RV) apex (white arrows). C, Three-dimensional TEE confirmed the obliteration of RV apex (red arrow) and showed a prominent aneurysm of fossa ovalis (white arrow). D and E, Myocardial contrast echocardiography evidenced a subtle left ventricular (LV) apex and marked RV apex subendocardial delayed perfusion associated with a perfusion defect over the RV endocardium (white arrows). F, CMR showed RV apical thickening and obliteration, typical late double V enhancement compatible with subendocardial fibrosis and thrombus, besides the presence of LV late subendocardial enhancement in its apex and apical lateral segment (white arrows).
The Data Supplement is available at https://www.ahajournals.org/doi/suppl/10.1161/CIRCIMAGING.120.012093.
Arquivos Brasileiros de Cardiologia
PMID: 13933908
ENDOMYOCARDIAL FIBROSIS. A CASE REPORT.
DEMATTOS AG, ACHUTTI A, FAGUNDES LA, DELIMA CP, FARACO E.
Cor Vasa. 1964;6:76-80. PMID: 14134507
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